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dc.contributor.authorBesnier, Francois
dc.contributor.authorSolberg, Monica Favnebøe
dc.contributor.authorHarvey, Alison C.
dc.contributor.authorCarvalho, Gary R.
dc.contributor.authorBekkevold, Dorte
dc.contributor.authorTaylor, Martin I.
dc.contributor.authorCreer, Simon
dc.contributor.authorNielsen, Einar Eg
dc.contributor.authorSkaala, Øystein
dc.contributor.authorAyllon, Fernando
dc.contributor.authorDahle, Geir
dc.contributor.authorGlover, Kevin
dc.date.accessioned2020-10-16T11:39:49Z
dc.date.available2020-10-16T11:39:49Z
dc.date.created2020-05-20T14:49:05Z
dc.date.issued2020
dc.identifier.citationBMC Genetics. 2020, 21 (1), .en_US
dc.identifier.issn1471-2156
dc.identifier.urihttps://hdl.handle.net/11250/2683336
dc.description.abstractQuantitative traits are typically considered to be under additive genetic control. Although there are indications that non-additive factors have the potential to contribute to trait variation, experimental demonstration remains scarce. Here, we investigated the genetic basis of growth in Atlantic salmon by exploiting the high level of genetic diversity and trait expression among domesticated, hybrid and wild populations.en_US
dc.language.isoengen_US
dc.titleEpistatic regulation of growth in Atlantic salmon revealed: a QTL study performed on the domesticated-wild interfaceen_US
dc.typePeer revieweden_US
dc.typeJournal articleen_US
dc.description.versionpublishedVersionen_US
dc.source.pagenumber17en_US
dc.source.volume21en_US
dc.source.journalBMC Geneticsen_US
dc.source.issue1en_US
dc.identifier.doi10.1186/s12863-020-0816-y
dc.identifier.cristin1811955
dc.relation.projectNorges forskningsråd: 200510en_US
dc.relation.projectNorges forskningsråd: 216105en_US
dc.relation.projectEC/FP7/311920en_US
cristin.ispublishedtrue
cristin.fulltextoriginal
cristin.qualitycode1


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